Author

A. G. Elefanty

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

B. Reljic· 1 shared C. Bonifer· 1 shared D. L. Vaux· 1 shared E. G. Stanley· 1 shared E. Ng· 1 shared E. S. Ng· 1 shared H. Tran· 1 shared J. G. Cheung· 1 shared

Activity by Year

2018-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

B. Reljic

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 28, 2019

RUNX1-ETO induction rapidly alters chromatin landscape and growth of a specific sub-population of hESC-derived myeloid precursor cells by interfering with RUNX1 regulation

M. Nafria, P. Keane, E. Ng…·C. Bonifer

This study shows that RUNX1-ETO expression in hESC-derived myeloid precursors causes chromatin reprogramming, blocks differentiation, and arrests growth, with effects specific to a sub-population of cells.

DOI 10.1101/748921

humanChIP-seq

𝕏 PostBluesky

Cell BiologybioRxivNov 15, 2018

In the absence of apoptosis, myeloid cells arrest when deprived of growth factor, but remain viable by consuming extracellular glucose

L. Dong, B. Reljic, J. G. Cheung…·H. Tran

The paper shows that Bax-/-Bak1-/- myeloid cells deprived of IL3 survive by consuming extracellular glucose, without needing Atg5-dependent autophagy.

DOI 10.1038/s41418-019-0275-z

mousebulk RNA-seq

𝕏 PostBluesky