Author

A. G. Kotini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

A. R. Pine· 1 shared C. S. Leslie· 1 shared E. P. Papapetrou· 1 shared H. Yuan· 1 shared J. Wesely· 1 shared L. Zamparo· 1 shared T. Wang· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. R. Pine

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Sequential CRISPR gene editing in human iPSCs charts the clonal evolution of leukemia

T. Wang, A. R. Pine, J. Wesely, H. Yuan, L. Zamparo, A. G. Kotini, C. S. Leslie, E. P. Papapetrou

This study uses sequential CRISPR gene editing in human iPSCs to model clonal evolution in AML, showing progressive dysplasia and identifying therapeutic vulnerabilities.

Cell Stem Cell · Jun 1, 2021Peer-reviewedCancer Biology