Author

A. H. V. Schapira

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. V. Bradshaw· 1 shared C. Galvagnion· 1 shared D. A. Di Monte· 1 shared F. Blandini· 1 shared H. R. Morris· 1 shared J.-W. Taanman· 1 shared P. Campbell· 1 shared S. Cerri· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 12 mutual co-author links

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1 shared paperCell Biology

A. V. Bradshaw

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2 papers

Recent·Most cited

BiochemistryBrainPeer-reviewedApr 1, 2022

Sphingolipid changes in Parkinson L444P GBA mutation fibroblasts promote α-synuclein aggregation

C. Galvagnion, S. Cerri, A. H. V. Schapira…·D. A. Di Monte

This study shows that the L444P GBA mutation in fibroblasts alters sphingolipid profiles, promoting α-synuclein aggregation through lipid-induced fibrillation.

DOI 10.1093/brain/awab371

humanmetabolomics

𝕏 PostBluesky

Cell BiologybioRxivFeb 12, 2020

The PINK1 - Parkin mitophagy signalling pathway is not functional in peripheral blood mononuclear cells

A. V. Bradshaw, P. Campbell, A. H. V. Schapira…·J.-W. Taanman

The study shows that the PINK1-Parkin mitophagy pathway is not functional in peripheral blood mononuclear cells, making them unsuitable for mitophagy analysis in Parkinson's disease.

DOI 10.1371/journal.pone.0259903

humanproteomics

𝕏 PostBluesky