Author

A. Hirao

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Organism:Arabidopsis· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1

Frequent Co-Authors

E. Hinoi· 1 shared G. Park· 1 shared H. Sabit· 1 shared K. Fukasawa· 1 shared K. Kaneda· 1 shared K. Shimizu· 1 shared K. Tokumura· 1 shared M. Hiraiwa· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

E. Hinoi

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

SMURF2 phosphorylation at Thr249 modifies the stemness and tumorigenicity of glioma stem cells by regulating TGF-β receptor stability

M. Hiraiwa, K. Fukasawa, T. Iezaki, H. Sabit, T. Horie, K. Tokumura, S. Iwahashi, M. Murata, M. Kobayashi, G. Park, K. Kaneda, T. Todo, A. Hirao, M. Nakada, E. Hinoi

This study shows that SMURF2 phosphorylation at Thr249 regulates GSC stemness and tumorigenicity by affecting TGF-β receptor stability.

bioRxiv · Apr 27, 2021Cancer Biology

Experimental and field data support habitat expansion of the allopolyploid Arabidopsis kamchatica owing to parental legacy of heavy metal hyperaccumulation

T. Paape, R. Akiyama, T. Cereghetti, Y. Onda, A. Hirao, T. Kenta, K. Shimizu

The paper shows that the allopolyploid Arabidopsis kamchatica retains hyperaccumulation traits from its parent A. halleri, enabling it to expand into heavy metal-rich habitats.

bioRxiv · Oct 21, 2019Evolutionary Biology