Author

A. L. Cadell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

D. R. Croucher· 2 shared P. D. Mace· 2 shared A. E. Burgess· 1 shared B. L. Parker· 1 shared C. L. Day· 1 shared J. L. Brewster· 1 shared J. M. Crowther· 1 shared J. M. Murphy· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBiochemistry

D. R. Croucher

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A cryptic tubulin-binding domain links MEKK1 to microtubule remodelling

P. Filipcik, S. L. Latham, A. L. Cadell, C. L. Day, D. R. Croucher, P. D. Mace

This paper identifies a previously unknown TOG domain in MEKK1 that binds curved tubulin heterodimers, and shows mutations disrupting this interaction are enriched in tumors.

bioRxiv · Apr 7, 2020 · PreprintBiochemistry

Mechanism of preferential complex formation by Apoptosis Signal-regulating Kinases

S. J. Trevelyan, J. L. Brewster, A. E. Burgess, J. M. Crowther, A. L. Cadell, B. L. Parker, D. R. Croucher, R. C. Dobson, J. M. Murphy, P. D. Mace

This study identifies the mechanism by which ASK1-3 kinases form preferential heterocomplexes through their SAM domains, revealing structural insights into their role in inflammatory signaling.

bioRxiv · Jul 5, 2019Biochemistry