Author

A. Namdar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 1 Organism:mouse· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

C. Mowat· 1 shared D. Schiller· 1 shared G. Dunsmore· 1 shared J. Jovel· 1 shared K. Baker· 1 shared P. Koleva· 1 shared S. Elahi· 1 shared S. Houston· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

C. Mowat

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Antitumor immunity in dMMR colorectal cancers requires interferon-induced CCL5 and CXCL10

C. Mowat, S. R. Mosley, A. Namdar, D. Schiller, K. Baker

This study identifies that dMMR CRCs recruit CD8+ T cells via CCL5 and CXCL10 overexpression driven by cGAS/STING and IFN signaling, offering a therapeutic target for improving immune response in other CRCs.

bioRxiv · Sep 15, 2020Cancer Biology

CD71+ erythroid cells exacerbate HIV-1 infection by reactive oxygen species and trans-infect HIV to CD4+ T cells

S. Elahi, A. Namdar, G. Dunsmore, P. Koleva, S. Shahbaz, J. Jovel, S. Houston

This study shows CD71+ erythroid cells enhance HIV-1 infection through reactive oxygen species and trans-infect CD4+ T cells, even in the presence of antiretroviral drugs.

bioRxiv · Apr 29, 2019 · PreprintImmunology