Author

A. S. Bais

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

B. J. Aronow· 1 shared C. K. Kuo· 1 shared C. W. Lo· 1 shared D. Kostka· 1 shared G. A. Porter· 1 shared G. Beutner· 1 shared G. S. Pryhuber· 1 shared H. Yagi· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

B. J. Aronow

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

iPSC modeling shows uncompensated mitochondrial mediated oxidative stress underlies early heart failure in hypoplastic left heart syndrome

X. Xu, K. Jin, A. S. Bais, W. Zhu, H. Yagi, T. N. Feinstein, P. Nguyen, J. Criscione, X. Liu, G. Beutner, K. B. Karunakaran, P. Adams, C. K. Kuo, D. Kostka, G. S. Pryhuber, , C. W. Lo

This study uses iPSC-derived cardiomyocytes to show that mitochondrial oxidative stress and ER stress underlie early heart failure in HLHS patients.

bioRxiv · May 9, 2021 · PreprintCell Biology