Author

B. J. Aronow

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Assay:scRNA-seq· 3 Research Focus:cell-cell communication· 2 Approach:deep learning· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

K. Jin· 3 shared A. Mihalas· 1 shared A. Mitelpunkt· 1 shared A. Patel· 1 shared A. S. Bais· 1 shared C. E. Cucinotta· 1 shared C. J. Plaisier· 1 shared C. K. Kuo· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersCancer Biology

K. Jin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

KAT5 regulates neurodevelopmental states associated with G0-like populations in glioblastoma

A. Mihalas, S. Arora, S. O'Connor, H. Feldman, C. E. Cucinotta, K. Mitchell, J. Bassett, D. Kim, K. Jin, P. Hoellerbauer, J. Delegard, M. Ling, W. Jenkins, M. Kufeld, P. Corrin, , P. PADDISON

This study identifies KAT5 as a regulator of G0-like states in glioblastoma, showing its role in suppressing non-dividing subpopulations and associating with worse patient outcomes.

bioRxiv · Mar 17, 2022Cancer Biology

Implicating Gene and Cell Networks Responsible for Differential COVID-19 Host Responses via an Interactive Single Cell Web Portal

K. Jin, E. E. Bardes, A. Mitelpunkt, Y. J. Wang, S. Bhatnagar, S. Sengupta, D. P. Krummel, M. E. Rothenberg, B. J. Aronow

The paper creates a web portal to compare single-cell transcriptome data from COVID-19 and control samples, identifying gene and cell network hypotheses related to differential host responses.

bioRxiv · Jun 7, 2021Immunology

iPSC modeling shows uncompensated mitochondrial mediated oxidative stress underlies early heart failure in hypoplastic left heart syndrome

X. Xu, K. Jin, A. S. Bais, W. Zhu, H. Yagi, T. N. Feinstein, P. Nguyen, J. Criscione, X. Liu, G. Beutner, K. B. Karunakaran, P. Adams, C. K. Kuo, D. Kostka, G. S. Pryhuber, , C. W. Lo

This study uses iPSC-derived cardiomyocytes to show that mitochondrial oxidative stress and ER stress underlie early heart failure in HLHS patients.

bioRxiv · May 9, 2021 · PreprintCell Biology