Author

A. Tiwaa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:clinical cohort / trial· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

C. L. Simpson· 2 shared J. E. Gudjonsson· 2 shared B. C. Capell· 1 shared C. J. Johnson· 1 shared E. Y. Chu· 1 shared J. Zou· 1 shared M. K. Sarkar· 1 shared P. W. Harms· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

C. L. Simpson

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2 papers

Recent·Most cited

ERK hyperactivation in epidermal keratinocytes impairs intercellular adhesion and drives Grover disease pathology

C. L. Simpson, A. Tiwaa, S. A. Zaver, C. J. Johnson, E. Y. Chu, P. W. Harms, J. E. Gudjonsson

This study shows ERK hyperactivation in keratinocytes causes impaired adhesion and drives Grover disease, supporting MEK inhibition as a potential therapy.

bioRxiv · Apr 30, 2024Cell Biology

Targeting SERCA2 in organotypic epidermis reveals MEK inhibition as a therapeutic strategy for Darier disease.

S. A. A. Zaver, M. K. Sarkar, S. Egolf, J. Zou, A. Tiwaa, B. C. Capell, J. E. Gudjonsson, C. L. Simpson

This study uses human organotypic epidermis to show that SERCA2 deficiency disrupts keratinocyte adhesion via ERK signaling, and identifies MEK inhibition as a potential therapy for Darier disease.

bioRxiv · Mar 7, 2023Cell Biology