Author

Aileen Ning

University of California, Los Angeles

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:human· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

Bianca E. RusselliD· 2 shared Isabella LiniD· 2 shared Valerie A. ArboledaiD· 2 shared Abigail S. Krall· 1 shared Analeyla Herrera· 1 shared Angela WeiiD· 1 shared Heather R. ChristofkiD· 1 shared Linsey StilesiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetics

Bianca E. RussellORCID

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2 papers

Recent·Most cited

Truncating ASXL1 variants rewire cellular metabolism via mitochondrial pyruvate carrier repression

Isabella Lin, Michael Sigfrid S. Reyes, Abigail S. Krall, Neerja Vashist, Suzanna Sarkissian, Nedas Matulionis, Aileen Ning, Linsey Stiles, Bianca E. Russell, Paul Mark B. Medina, Heather R. Christofk, Valerie A. Arboleda

This paper shows that truncating ASXL1 variants disrupt mitochondrial pyruvate transport, leading to metabolic changes and Wnt signaling dysregulation in Bohring-Opitz syndrome and myeloid leukemia.

bioRxiv · Jul 17, 2026 · PreprintGenetics

Truncating ASXL1 mutations in Bohring-Opitz Syndrome dysregulate canonical and non-canonical Wnt Signaling

Isabella Lin, Angela Wei, Zain Awamleh, Meghna Singh, Aileen Ning, Analeyla Herrera, Bianca E. Russell, Rosanna Weksberg, Valerie A. Arboleda

The paper identifies how ASXL1 mutations disrupt the epigenome and activate Wnt signaling pathways in Bohring-Opitz syndrome and myeloid leukemias.

bioRxiv · Dec 15, 2022 · PreprintGenetics