Author

Alexa Rinsant

Centre Hospitalier Universitaire de Poitiers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:protein structure· 1

Frequent Co-Authors

Alessio LampisiD· 1 shared Alexander CarpinteiroiD· 1 shared Amélie BonaudiD· 1 shared Arnaud JaccardiD· 1 shared Cécile Ory· 1 shared Christelle ObletiD· 1 shared Christophe SiraciD· 1 shared Diana CanettiiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPathology

Alessio LampisORCID

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1 paper

Recent·Most cited

A mouse model of cardiac AL amyloidosis unveils mechanisms of tissue accumulation and toxicity of amyloid fibrils

Gemma Martinez-Rivas, Maria Victoria Ayala, Sébastien Bender, Gilles Roussine Codo, Karolina Weronika Świderska, Alessio Lampis, Laura Pedroza, Melisa Merdanovic, Pierre Sicard, Émilie Pinault, Laurence Richard, Francesca Lavatelli, Sofia Giorgetti, Diana Canetti, Alexa Rinsant, , Christophe Sirac

This paper develops a mouse model of cardiac AL amyloidosis that reproduces human disease features, including amyloid deposits and cardiac dysfunction, and identifies a role for partial LC degradation in fibril formation.

Nature Communications · Mar 27, 2025Peer-reviewedPathology