Author

Arnaud Jaccard

Centre National de la Recherche ScientifiqueORCID 0000-0001-7091-8253Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:protein structure· 1

Frequent Co-Authors

Christophe SiraciD· 2 shared Frank BridouxiD· 2 shared Gemma Martinez-RivasiD· 2 shared Sébastien BenderiD· 2 shared Alan C. StuckeyiD· 1 shared Alessio LampisiD· 1 shared Alexa Rinsant· 1 shared Alexander CarpinteiroiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPathology

Christophe SiracORCID

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2 papers

Recent·Most cited

PathologybioRxivPreprintDec 25, 2025

Preclinical characterization of AT-03, a novel Serum Amyloid P fusion protein that demonstrates pan-amyloid binding and removal

Christophe Sirac, Gilles Roussine Codo, Arnaud Jaccard…·Jonathan S. Wall

This paper characterizes AT-03, a novel fusion protein that binds and removes various amyloid types, showing therapeutic efficacy in a mouse model.

1 citation

mouseexperimental assay

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PathologyNature CommunicationsPeer-reviewedMar 27, 2025

A mouse model of cardiac AL amyloidosis unveils mechanisms of tissue accumulation and toxicity of amyloid fibrils

Gemma Martinez-Rivas, Maria Victoria Ayala, Sébastien Bender…·Christophe Sirac

This paper develops a mouse model of cardiac AL amyloidosis that reproduces human disease features, including amyloid deposits and cardiac dysfunction, and identifies a role for partial LC degradation in fibril formation.

14 citationstop 1%

mouseprotein structure

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