Author

B. Dagg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

B. Khatri· 2 shared M. M. Ho· 2 shared A. Rodgers· 1 shared D. A. Kaveh· 1 shared D. Evangelopoulos· 1 shared G. Prosser· 1 shared J. Keeble· 1 shared L. P. Carvalho· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersImmunology

B. Khatri

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2 papers

Recent·Most cited

Efficacy and immunogenicity of different BCG doses in BALB/c and CB6F1 mice when challenged with H37Rv or Beijing HN878

B. Khatri, J. Keeble, B. Dagg, D. A. Kaveh, P. J. Hogarth, M. M. Ho

This study evaluates BCG vaccine efficacy and immunogenicity in mice at different doses against two Mycobacterium tuberculosis strains, finding protection and immune responses at specific dose ranges.

bioRxiv · Oct 21, 2020Immunology

Antibiotic resistance evasion is explained by rare mutation frequency and not by lack of compensatory mechanisms

L. P. Carvalho, D. Evangelopoulos, G. Prosser, A. Rodgers, B. Dagg, B. Khatri, M. M. Ho, M. Gutierrez, T. Cortes

The study shows that Mycobacterium tuberculosis does not develop resistance to D-cycloserine due to its ultra-low mutation frequency, not because of a lack of compensatory mechanisms.

bioRxiv · Jul 23, 2018 · PreprintMicrobiology