Author

Benjamin Ober‐Reynolds

Stanford Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:scATAC-seq· 2 Assay:scRNA-seq· 2 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Approach:experimental assay· 1

Frequent Co-Authors

William James GreenleafiD· 2 shared Amy F. CheniD· 1 shared Anthony E. OroiD· 1 shared Arwa S. Kathiria· 1 shared Benjamin E. ParksiD· 1 shared Chen WangiD· 1 shared Eon J. Rios· 1 shared Jörg J. GoronzyiD· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersGenomics

William James GreenleafORCID

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2 papers

Recent·Most cited

GenomicsbioRxivPreprintSep 28, 2022

Integrated single-cell chromatin and transcriptomic analyses of human scalp reveal etiological insights into genetic risk for hair and skin disease

Benjamin Ober‐Reynolds, Chen Wang, Justin Ko…·William James Greenleaf

The paper integrates single-cell chromatin and transcriptomic data from human scalp to identify gene regulatory mechanisms and causal variants in hair and skin diseases.

DOI 10.1101/2022.09.28.509620

humanscRNA-seq

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GenomicsNature MethodsPeer-reviewedMay 1, 2022

NEAT-seq: Simultaneous profiling of intra-nuclear proteins, chromatin accessibility, and gene expression in single cells

Amy F. Chen, Benjamin E. Parks, Arwa S. Kathiria…·William James Greenleaf

NEAT-seq profiles nuclear proteins, chromatin accessibility, and transcriptomes in single cells, revealing TF regulatory mechanisms and linking GWAS SNPs to chromatin accessibility and target genes.

139 citationstop 1%

humanscRNA-seq

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