Author

C. Megli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Organism:mouse· 1

Frequent Co-Authors

A. L. Hartman· 1 shared A. McElroy· 1 shared B. Bird· 1 shared C. B. Coyne· 1 shared C. McMillen· 1 shared C. Ovies· 1 shared D. A. Boyles· 1 shared E. C. Semmes· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperMicrobiology

A. L. Hartman

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Vaccine strains of Rift Valley fever virus exhibit attenuation at the maternal-fetal placental interface

C. McMillen, C. Megli, R. Radisic, L. Skvarca, R. M. Hoehl, D. A. Boyles, J. J. McGaughey, B. Bird, A. McElroy, A. L. Hartman

This study shows that vaccine strains of Rift Valley fever virus replicate less in placental tissues compared to wild-type strains, suggesting attenuation at the maternal-fetal interface.

bioRxiv · May 31, 2024Microbiology

Bi-directional communication between monocytes and trophoblasts under hypoxia and hypoxia-reperfusion conditions

H. Yankello, Y. Lee, C. Megli, E. C. Wayne

This study examines how hypoxia and hypoxia-reperfusion conditions in placental cells affect trophoblast-monocyte interactions and monocyte function.

bioRxiv · Sep 23, 2023 · PreprintBioengineering

Innate immune signaling in trophoblast and decidua organoids defines differential antiviral defenses at the maternal-fetal interface

L. Yang, E. C. Semmes, C. Ovies, C. Megli, S. Permar, J. B. Gilner, C. B. Coyne

The paper develops trophoblast and decidua organoids to study antiviral defenses at the maternal-fetal interface, showing differential innate immune responses and susceptibility to HCMV.

bioRxiv · Mar 29, 2021Immunology