Author

C. Munoz-Fontela

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

B. E. Nilsson-Payant· 1 shared B. Escudero· 1 shared B. Lee· 1 shared B. tenOever· 1 shared C. Krisp· 1 shared C. Uetrecht· 1 shared D. Blanco-Melo· 1 shared G. Allmaier· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperMicrobiology

B. E. Nilsson-Payant

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2 papers

Recent·Most cited

BiophysicsbioRxivNov 25, 2020

N-terminal VP1 truncations favor T=1 norovirus-like particles

R. Pogan, V. U. Weiss, K. Bond…·C. Uetrecht

The paper identifies N-terminal VP1 truncations as a cause for T=1 norovirus-like particle formation and discusses implications for vaccine and biotechnological applications.

DOI 10.3390/vaccines9010008

humanproteomics

𝕏 PostBluesky

MicrobiologybioRxivPreprintMar 12, 2020

An inability to maintain the ribonucleoprotein genomic structure is responsible for host detection of negative-sense RNA viruses

D. Blanco-Melo, B. E. Nilsson-Payant, S. Uhl…·B. tenOever

The paper shows that limiting nucleoprotein (NP) in negative-sense RNA viruses leads to defective genomes and triggers host interferon responses, making NP a key antiviral target.

DOI 10.1101/2020.03.12.989319

humanexperimental assay

𝕏 PostBluesky