Author

C. Pileggi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

M.-E. Harper· 2 shared A. Atrih· 1 shared A. Beauvais· 1 shared A. Tierney· 1 shared A.-A. Dumont· 1 shared B. L. Schneider· 1 shared D. Lamont· 1 shared D. P. Blondin· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPhysiology

M.-E. Harper

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Author profile →

2 papers

Recent·Most cited

Cardiomyocyte-specific Srsf3 deletion reveals a mitochondrial regulatory role

A.-A. Dumont, L. Dumont, D. Zhou, H. Giguere, C. Pileggi, M.-E. Harper, D. P. Blondin, M. S. Scott, M. Auger-Messier

This study shows that deleting Srsf3 in cardiomyocytes leads to mitochondrial dysfunction, reduced survival, and cardiac remodeling in mice.

bioRxiv · Jul 3, 2020Physiology

A mouse model for spinal muscular atrophy provides insights into non-alcoholic fatty liver disease pathogenesis

M.-O. Deguise, C. Pileggi, A. Beauvais, A. Tierney, L. Chehade, Y. De Repentigny, J. Michaud, M. Llavero-Hurtado, D. Lamont, A. Atrih, T. M. Wishart, T. H. Gillingwater, B. L. Schneider, M.-E. Harper, S. H. Parson, R. Kothary

This paper uses a mouse model of spinal muscular atrophy to study non-alcoholic fatty liver disease, finding that it develops steatohepatitis and identifies molecular mechanisms involved.

bioRxiv · Apr 29, 2020 · PreprintPathology