Author

M. Auger-Messier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

E. Marsault· 2 shared H. Giguere· 2 shared M. S. Scott· 2 shared A. Desroches· 1 shared A.-A. Dumont· 1 shared C. F. Bentzinger· 1 shared C. Pileggi· 1 shared D. P. Blondin· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

E. Marsault

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3 papers

Recent·Most cited

Apelin improves angiogenesis and blood flow reperfusion following lower limb ischemia in diabetic mice

S. Robillard, K. Tran, T. Brazeau, E. Boisvert, F. Lizotte, M. Auger-Messier, P.-L. Boudreault, E. Marsault, P. Geraldes

This study shows that Pyr-apelin-13 improves blood flow and angiogenesis in diabetic mice with limb ischemia, overcoming diabetes-induced resistance to VEGF.

bioRxiv · Feb 8, 2023 · PreprintCell Biology

Apelin Stimulation of the Perivascular MuSC Niche Enhances Endogenous Repair in Muscular Dystrophy

E. Le Moal, Y. Liu, J. Collerette-Tremblay, S. Dumontier, J. Boutin, J. Dort, Z. Orfi, J. Michaud, H. Giguere, A. Desroches, F. Vezina, S. Bedard, F. Balg, P. Sarret, M. S. Scott, , C. F. Bentzinger

This study shows that apelin-13 enhances muscle regeneration in dystrophic mice by stimulating endothelial cells in the perivascular niche, improving MuSC function and tissue repair.

bioRxiv · Sep 9, 2022Cell Biology

Cardiomyocyte-specific Srsf3 deletion reveals a mitochondrial regulatory role

A.-A. Dumont, L. Dumont, D. Zhou, H. Giguere, C. Pileggi, M.-E. Harper, D. P. Blondin, M. S. Scott, M. Auger-Messier

This study shows that deleting Srsf3 in cardiomyocytes leads to mitochondrial dysfunction, reduced survival, and cardiac remodeling in mice.

bioRxiv · Jul 3, 2020Physiology