Author

C. R. Walkley

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. E. Frazier· 1 shared A. M. Chalk· 1 shared J. J. Xu· 1 shared M. F. Smeets· 1 shared M. Smeets· 1 shared M. Wall· 1 shared N. A. Sims· 1 shared W. Castillo-Tandazo· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 16 mutual co-author links

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1 shared paperCancer Biology

A. E. Frazier

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2 papers

Recent·Most cited

Srsf2P95H/+ co-operates with loss of TET2 to promote myeloid bias and initiate a chronic myelomonocytic leukemia like disease in mice.

J. J. Xu, A. M. Chalk, M. Wall, W. Y. Langdon, M. F. Smeets, C. R. Walkley

This paper shows that Srsf2P95H/+ and Tet2 loss cooperate in mice to cause a CMML-like disease with myeloid bias and monocytosis.

bioRxiv · Jun 21, 2022Cancer Biology

Rothmund-Thomson Syndrome-like RECQL4 truncating mutations cause a haploinsufficient low bone mass phenotype in mice

W. Castillo-Tandazo, A. E. Frazier, N. A. Sims, M. Smeets, C. R. Walkley

This study shows that truncating RECQL4 mutations in mice cause low bone mass and reduced cortical bone thickness, highlighting RECQL4 gene dosage as a regulator of bone mass.

bioRxiv · Nov 11, 2020Cancer Biology