Author

Carl Koschmann

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Sriram VennetiiD· 2 shared Alan Meeker· 1 shared Anda-Alexandra Calinescu· 1 shared Anne Marie GillespieiD· 1 shared Cameron J. Herting· 1 shared Carlen baker· 1 shared Céline TaglangiD· 1 shared Costas A. Lyssiotis· 1 shared

Activity by Year

2018-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

Sriram VennetiORCID

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Author profile →

3 papers

Recent·Most cited

Oncohistone inhibition reshapes tumor-microenvironment communication in Diffuse Midline Glioma (DMG)

Niloofar Khairkhah, Mostafa M.H. Ibrahim, Sienna L. Galban, Megan Faunce, Lily Rober, Carlen baker, Robert Doherty, Rodrigo Cartaxo, Carl Koschmann, Yue Zhao, Stefanie Galban

This study shows that H3K27M oncohistone reshapes the tumor microenvironment in DMG by altering neuronal interactions and immune signaling, suggesting it as a therapeutic target.

bioRxiv · Sep 16, 2026 · PreprintCancer Biology

Lactylation fuels nucleotide biosynthesis and facilitates deuterium metabolic imaging of tumor proliferation in H3K27M-mutant gliomas

Georgios Batsios, Céline Taglang, Suresh Udutha, Anne Marie Gillespie, Simon P. Robinson, Timothy Phoenix, Sabine Mueller, Sriram Venneti, Carl Koschmann, Pavithra Viswanath

This paper shows lactate supports nucleotide biosynthesis in H3K27M-mutant gliomas and enables deuterium metabolic imaging of tumor proliferation.

Science Translational Medicine · Feb 11, 2026Peer-reviewedCancer Biology

IDH1R132H acts as a tumor suppressor in glioma via epigenetic upregulation of the DNA damage response

Felipe J. Núñez, Flor M. Mendez, Padma Kadiyala, Mahmoud S. Alghamri, Masha G. Savelieff, Carl Koschmann, Anda-Alexandra Calinescu, Neha Kamran, Rohin Patel, Stephen Carney, Marissa Z. Guo, Maria B. Garcia-Fabiani, Santiago Haase, Marta Edwards, Mats Ljungman, , Maria G. Castro

The paper shows IDH1R132H mutation in glioma improves survival and DNA repair by enhancing ATM signaling and radioresistance, which can be reversed by inhibiting ATM or CHK1/2.

bioRxiv · Aug 10, 2018 · PreprintCancer Biology