Author

Cody Zurhellen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Caleb A. WoodiD· 1 shared Esther L. Dela Cruz· 1 shared George A. EdwardsiD· 1 shared Hannah L. Dela Cruz· 1 shared Herbert T. York· 1 shared Ismael Al‐RamahiiD· 1 shared Jay S. Dela Cruz· 1 shared Jim A. Baun· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Caleb A. WoodORCID

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2 papers

Recent·Most cited

TMEM106B coding variant is protective and deletion detrimental in a mouse model of tauopathy

George A. Edwards, Caleb A. Wood, Yang He, Quynh Nguyen, Peter J. Kim, Ruben Gomez‐Gutierrez, Kyung-Won Park, Yong Xu, Cody Zurhellen, Ismael Al‐Ramahi, Joanna L. Jankowsky

This study shows that TMEM106B deletion worsens tauopathy symptoms in mice, while a coding variant provides protection against cognitive decline and neurodegeneration.

Acta Neuropathologica · Mar 25, 2024Peer-reviewedNeuroscience

New insights underlying the early events of dopaminergic dysfunction in Parkinson's Disease

Hannah L. Dela Cruz, Esther L. Dela Cruz, Cody Zurhellen, Herbert T. York, Jim A. Baun, Joshua L. Dela Cruz, Jay S. Dela Cruz

This paper shows that elevated alpha-MSH in Parkinson's disease models impairs autophagy, leading to alpha-synuclein accumulation and motor deficits, which can be temporarily reversed by dopamine replacement.

bioRxiv · Sep 27, 2020 · PreprintNeuroscience