Author

D. B. Davis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Biochemistry· 1 Cell Biology· 1 Pharmacology & Toxicology· 1

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Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:electrophysiology· 1 Organism:mouse· 1

Frequent Co-Authors

A. H. de Souza· 1 shared A. L. Brill· 1 shared A. M. Weeks· 1 shared A. Reuter· 1 shared B. Wancewicz· 1 shared C. E. Kaiser· 1 shared C. Patibandla· 1 shared C. R. Kibbe· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. H. de Souza

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3 papers

Recent·Most cited

A plasma membrane-associated glycolytic metabolon is functionally coupled to KATP channels in pancreatic α and β cells from humans and mice

T. Ho, E. Potapenko, D. B. Davis, M. J. Merrins

The paper demonstrates a glycolytic metabolon on the plasma membrane that directly controls KATP channels in pancreatic α and β cells from humans and mice through ATP/ADP and NAD+/NADH cycles.

bioRxiv · Oct 6, 2022 · PreprintBiochemistry

Cholecystokinin Suppresses β-Cell Apoptosis, Including in Human Islets in a Transplant Model

H. T. Kim, A. H. de Souza, H. Umhoefer, J. Han, L. Anzia, S. J. Sacotte, R. A. Williams, J. T. Blumer, J. T. Bartosiak, D. A. Fontaine, M. Baan, C. R. Kibbe, D. B. Davis

This study shows that cholecystokinin reduces β-cell apoptosis in human islets both in vitro and in a transplant model, suggesting its therapeutic potential for diabetes.

bioRxiv · Mar 2, 2021 · PreprintCell Biology

Human islet expression levels of Prostaglandin E2 synthetic enzymes, but not prostaglandin EP3 receptor, are positively correlated with markers of β-cell function and mass in non-diabetic obesity

N. A. Truchan, R. J. Fenske, H. K. Sandhu, A. M. Weeks, C. Patibandla, B. Wancewicz, S. Pabich, A. Reuter, J. M. Harrington, A. L. Brill, D. C. Peter, R. Nall, M. Daniels, M. Punt, C. E. Kaiser, , M. E. Kimple

The paper examines the relationship between PGE2 production and β-cell function in non-diabetic obesity, finding that PGE2 synthetic enzymes correlate with β-cell mass and function, while EP3 receptor does not.

bioRxiv · Feb 3, 2021Pharmacology & Toxicology