Author

D. Höper

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

M. Beer· 2 shared A. Breithaupt· 1 shared A. Godel· 1 shared A. Kratzel· 1 shared A. Rauch· 1 shared A. Taddeo· 1 shared B. Hoffmann· 1 shared B. I. O. Esteves· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersMicrobiology

M. Beer

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2 papers

Recent·Most cited

The absence of superinfection exclusion of Borna disease virus 2 maintains genomic polymorphisms in persistently infected cells

T. Kanda, P. D. Santos, D. Höper, M. Beer, D. Rubbenstroth, K. Tomonaga

This study identifies genetic variations in BoDV-2 that enhance its growth and reveals its ability to maintain genomic polymorphisms in infected cells by lacking superinfection exclusion.

bioRxiv · Apr 15, 2024 · PreprintMicrobiology

The spike gene is a major determinant for the SARS-CoV-2 Omicron-BA.1 phenotype

T. Barut, N. J. Halwe, A. Taddeo, J. N. Kelly, J. Schön, N. Ebert, L. Ulrich, C. Devisme, S. Steiner, B. S. Trueeb, B. Hoffmann, I. B. Veiga, N. F. G. Leborgne, E. A. Moreira, A. Breithaupt, , V. Thiel

This study shows the spike gene determines replication, pathogenicity, and immune evasion of SARS-CoV-2 Omicron-BA.1 compared to Delta in cell cultures and animal models.

bioRxiv · Apr 28, 2022Microbiology