Author

A. Rauch

University of Bern

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cell Biology· 1 Evolutionary Biology· 1 Genetic & Genomic Medicine· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 5 Research Focus:host-pathogen & immune response· 4 Approach:experimental assay· 3 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Assay:metabolomics· 1

Frequent Co-Authors

E. Bernasconi· 3 shared M. Battegay· 2 shared M. Cavassini· 2 shared P. Schmid· 2 shared S. Yerly· 2 shared T. Klimkait· 2 shared A. A. Shanbhag· 1 shared A. Anagnostopoulos· 1 shared

Activity by Year

2017-2022 · peak 4/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersImmunology

E. Bernasconi

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

GIP receptor reduces osteoclast activity and improves osteoblast survival by activating multiple signaling pathways

M. S. Hansen, K. Soe, L. L. Christensen, P. Fernandez-Guerra, N. W. Hansen, R. A. Wyatt, C. Martin, R. S. Hardy, T. L. Anderson, J. B. Olesen, S. Overgaard, B. Hartmann, M. M. Rosenkilde, M. Kassem, A. Rauch, , M. Frost

This study shows GIP reduces osteoclast activity and improves osteoblast survival through multiple signaling pathways, suggesting GIPR agonism could treat osteoporosis.

bioRxiv · Jul 2, 2022Cell Biology

Anti-chemokine antibodies after SARS-CoV-2 infection correlate with favorable disease course

J. Muri, V. Cecchinato, A. Cavalli, A. A. Shanbhag, M. Matkovic, M. Biggiogero, P. A. Maida, J. Moritz, C. Toscano, E. Ghovehoud, R. Furlan, F. Barbic, A. Voza, G. De Nadai, C. Cervia, , Franzetti-Pelland

The paper finds that anti-chemokine antibodies after SARS-CoV-2 infection correlate with favorable disease outcomes and predict absence of long COVID symptoms.

bioRxiv · May 23, 2022 · PreprintImmunology

The spike gene is a major determinant for the SARS-CoV-2 Omicron-BA.1 phenotype

T. Barut, N. J. Halwe, A. Taddeo, J. N. Kelly, J. Schön, N. Ebert, L. Ulrich, C. Devisme, S. Steiner, B. S. Trueeb, B. Hoffmann, I. B. Veiga, N. F. G. Leborgne, E. A. Moreira, A. Breithaupt, , V. Thiel

This study shows the spike gene determines replication, pathogenicity, and immune evasion of SARS-CoV-2 Omicron-BA.1 compared to Delta in cell cultures and animal models.

bioRxiv · Apr 28, 2022Microbiology

Increased CHIP Prevalence Amongst People Living with HIV

Alexander G. Bick, Konstantin Popadin, Christian W. Thorball, Md Mesbah Uddin, Markella V. Zanni, Bing Yu, Matthias Cavassini, Andri Rauch, Philip Tarr, Patrick Schmid, Enos Bernasconi, Huldrych F. Günthard, Peter Libby, Eric Boerwinkle, Paul J. McLaren, , S. Yerly

The paper investigates the prevalence of clonal hematopoiesis of indeterminate potential (CHIP) in people living with HIV and finds a higher prevalence compared to controls.

Scientific Reports · Jan 12, 2022Peer-reviewedGenetic & Genomic Medicine

Quantifying the impact of gut microbiota on inflammation and hypertensive organ damage

E. G. Avery, H. Bartolomaeus, A. Rauch, C.-Y. Chen, G. N'diaye, U. Loeber, T. U. Bartolomaeus, R. Fritsche-Guenther, A. F. Rodrigues, D. Tsvetkov, M. Todiras, J.-K. Park, L. Marko, A. Maifeld, M. Bader, , N. Wilck

This study quantifies how gut microbiota affects inflammation and organ damage in hypertension using germ-free and colonized mice, finding the kidney is more susceptible to microbial influence.

bioRxiv · Sep 17, 2021Microbiology

Dissecting HIV Virulence: Heritability Of Setpoint Viral Load, CD4+ T Cell Decline And Per-Parasite Pathogenicity

F. Bertels, A. Marzel, G. Leventhal, V. Mitov, J. Fellay, H. F. Gunthard, J. Boni, S. Yerly, T. Klimkait, V. Aubert, M. Battegay, A. Rauch, M. Cavassini, A. Calmy, E. Bernasconi, , Swiss HIV Cohort Study

The paper estimates heritability of HIV virulence traits using donor-recipient regression and phylogenetic models, finding moderate heritability for CD4+ T cell decline and per-parasite pathogenicity.

bioRxiv · Sep 8, 2017Evolutionary Biology