Author

D. L. Goncalves

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Cell Biology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1 Organism:yeast· 1

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K. A. Morano· 1 shared S. Peffer· 1 shared

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2024-2024 · peak 1/yr

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1 shared paperCell Biology

K. A. Morano

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Cytoplasmic redox imbalance in the thioredoxin system activates Hsf1 and results in hyperaccumulation of the sequestrase Hsp42 with misfolded proteins

D. L. Goncalves, S. Peffer, K. A. Morano

This study shows that disrupting the cytosolic thioredoxin system in yeast activates Hsf1 and causes Hsp42 to hyperaccumulate with misfolded proteins, leading to growth defects under oxidative stress.

Molecular Biology of the Cell · Apr 1, 2024Peer-reviewedCell Biology