Author

D. M. Walter

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Cancer Biology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

A. A. Gudiel· 1 shared A. C. Gladstein· 1 shared D. C. Wallace· 1 shared D. M. Feldser· 1 shared I. A. Asangani· 1 shared J. O. Acosta· 1 shared K. M. Adler· 1 shared K. R. Doerig· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. A. Gudiel

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1 paper

Recent·Most cited

Setd2 inactivation sensitizes lung adenocarcinoma to inhibitors of oxidative respiration and mTORC1 signaling

D. M. Walter, A. C. Gladstein, K. R. Doerig, R. Natesan, S. G. Baskaran, A. A. Gudiel, K. M. Adler, J. O. Acosta, D. C. Wallace, I. A. Asangani, D. M. Feldser

This study shows that SETD2 inactivation in lung adenocarcinoma leads to increased oxidative metabolism and mTORC1 activity, making these tumors sensitive to inhibitors of oxidative respiration and mTORC1.

bioRxiv · Oct 6, 2022 · PreprintCancer Biology