Author

D. R. Holmes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Plant Biology· 3

Signature Assays, Organisms & Methods

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Organism:Arabidopsis· 3 Approach:experimental assay· 3 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

J. Monaghan· 2 shared M. Bredow· 2 shared A. Johnson Dingee· 1 shared A. Thomson· 1 shared C. A. Tanney· 1 shared C. Zipfel· 1 shared D. Ciren· 1 shared F. El Kasmi· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPlant Biology

J. Monaghan

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Author profile →

3 papers

Recent·Most cited

A novel allele in the Arabidopsis thaliana MACPF protein CAD1 results in deregulated immune signaling

D. R. Holmes, M. Bredow, K. Thor, S. A. Pascetta, I. Sementchoukova, K. R. Siegel, C. Zipfel, J. Monaghan

This study identifies a novel CAD1 allele in Arabidopsis thaliana that causes deregulated immune signaling and autoimmunity symptoms.

Genetics · Feb 19, 2021Peer-reviewedPlant Biology

Phosphorylation-dependent sub-functionalization of the calcium-dependent protein kinase CPK28

M. Bredow, K. W. Bender, A. Johnson Dingee, D. R. Holmes, A. Thomson, D. Ciren, C. A. Tanney, K. E. Dunning, M. Trujillo, S. C. Huber, J. Monaghan

This study identifies a phosphorylation site in CPK28 that differentially regulates its function in immune homeostasis and stem elongation in Arabidopsis.

bioRxiv · Oct 16, 2020Plant Biology

A shortcut in forward genetics: concurrent discovery of mutant phenotype and causal mutation in Arabidopsis M2 families via MAD-mapping

D. R. Holmes, R. Morbitzer, M. Wunderlich, H. Sun, F. El Kasmi, K. Schneeberger, T. Lahaye

This paper introduces MAD-mapping to identify causal mutations in M2 families directly, bypassing the need for F2 populations in forward genetics.

bioRxiv · Jun 29, 2020 · PreprintPlant Biology