Author

D. Sanchez-Taltavull

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Approach:experimental assay· 2 Assay:proteomics· 2 Organism:human· 1 Assay:metagenomics· 1 Approach:statistical model· 1

Frequent Co-Authors

D. Stroka· 3 shared G. Beldi· 3 shared D. Candinas· 2 shared D. Spari· 2 shared A. Schmid· 1 shared B. Yilmaz· 1 shared C. Mooser· 1 shared D. Pereyra· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersImmunology

D. Stroka

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Released Bacterial ATP Shapes Local and Systemic Inflammation during Abdominal Sepsis

D. Spari, A. Schmid, D. Sanchez-Taltavull, S. Murugan, K. Keller, N. Ennaciri, L. Salm, D. Stroka, G. Beldi

This study investigates how bacteria release ATP during sepsis and shows that bacterial ATP suppresses local immune responses and exacerbates systemic inflammation through outer membrane vesicles.

bioRxiv · Mar 7, 2024Microbiology

Compartmentalization of intestinal bacteria by hepatic ILC3s prevents infections after surgery

M. O. Jakob, D. Sanchez-Taltavull, B. Yilmaz, T. Malinka, C. Mooser, D. Spari, L. A. Salm, K. Freiburghaus, D. Pereyra, S. Hapfelmeier, M. Masoodi, P. Starlinger, D. Stroka, F. Tschan, D. Candinas, , G. Beldi

The paper shows that intestinal bacteria cause surgical infections and that hepatic ILC3s prevent their translocation, highlighting the role of controlled commensalism in preventing post-surgical infections.

bioRxiv · Sep 19, 2019 · PreprintImmunology

Connexin-43 dependent ATP release mediates macrophage activation during peritonitis

M. Dosch, J. Zindel, F. Jebbawi, N. Melin, D. Sanchez-Taltavull, D. Stroka, D. Candinas, G. Beldi

This study shows that Connexin-43-mediated ATP release from macrophages via TLR-2 and -4 agonists promotes sepsis severity, and inhibiting this pathway improves survival in peritonitis.

bioRxiv · Sep 23, 2018 · PreprintImmunology