Author

Daniel Davis

University of Missouri

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:bacteria (other)· 1 Organism:Drosophila· 1 Organism:mouse· 1

Frequent Co-Authors

Bing ZhangiD· 2 shared Agata Koprowska· 1 shared Ammara Rehman· 1 shared Andrew B. Reams· 1 shared Constancia Z. Dominguez· 1 shared Cynthia RecendeziD· 1 shared Elizabeth C. Bryda· 1 shared Elizabeth M. Reis· 1 shared

Activity by Year

2017-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

Bing ZhangORCID

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Author profile →

3 papers

Recent·Most cited

Novel Gain of Function Mouse Model of KCNT1-Related Epilepsy

Sarah L. Olguin, Daniel Davis, Ammara Rehman, Sean W. Berg, Elizabeth Sahagún, Payton S. Oswalt, Mandar Patil, Paula D.M. Sullivan, Bing Zhang, Elizabeth C. Bryda, Roy Ben‐Shalom, Jill L. Silverman

The paper creates a novel mouse model of KCNT1-related epilepsy carrying the G269S variant and demonstrates its phenotypic effects in vitro and in vivo.

bioRxiv · Jul 22, 2025 · PreprintNeuroscience

Gene amplification mutations originate prior to selective stress in Acinetobacter baylyi

Jennifer A. Herrmann, Agata Koprowska, Tesa J. Winters, Nancy Villanueva, Victoria D. Nikityuk, Feini Pek, Elizabeth M. Reis, Constancia Z. Dominguez, Daniel Davis, Eric McPherson, Staci R. Rocco, Cynthia Recendez, Shyla M. Difuntorum, Kelly Faeth, Mario D. Lopez, , Andrew B. Reams

This study shows gene amplification mutations in Acinetobacter baylyi occur before selective stress, challenging the adaptive amplification theory.

G3 Genes Genomes Genetics · Dec 12, 2022Peer-reviewedGenetics

Novel Compensatory Mechanisms Enable the Mutant KCNT1 Channels to Induce Seizures

Salleh N. Ehaideb, Gentry T. Decker, Petrina Smith, Daniel Davis, Bing Zhang

The paper shows that gain-of-function KCNT1 mutations cause seizures by silencing inhibitory neurons and inducing compensatory plasticity in motoneurons in Drosophila.

bioRxiv · Oct 2, 2017 · PreprintNeuroscience