Author

E. K. Barnell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:variant calling· 2 Assay:whole-exome sequencing· 2 Assay:whole-genome sequencing· 2

Frequent Co-Authors

M. Griffith· 3 shared O. L. Griffith· 3 shared K. Krysiak· 2 shared K. M. Campbell· 2 shared Z. L. Skidmore· 2 shared A. A. Chaudhuri· 1 shared A. R. Barnell· 1 shared C. A. Miller· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersCancer Biology

M. Griffith

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Author profile →

3 papers

Recent·Most cited

Genomic and transcriptomic somatic alterations of hepatocellular carcinoma in non-cirrhotic livers

Z. L. Skidmore, J. Kunisaki, Y. Lin, K. C. Cotto, E. K. Barnell, J. Hundal, K. Krysiak, V. Magrini, L. Trani, J. R. Walker, R. Fulton, E. Brunt, C. A. Miller, R. K. Wilson, E. R. Mardis, , O. L. Griffith

This study identifies genomic and transcriptomic alterations in non-cirrhotic hepatocellular carcinoma, revealing recurrent mutations and structural variants that may contribute to tumorigenesis.

bioRxiv · Dec 14, 2021Cancer Biology

Stool-derived eukaryotic RNA biomarkers for detection of high-risk adenomas

E. K. Barnell, Y. Kang, A. R. Barnell, K. M. Campbell, K. R. Kruse, E. M. Wurtzler, M. Griffith, A. A. Chaudhuri, O. L. Griffith

This study develops a stool-derived eukaryotic RNA biomarker model to detect high-risk adenomas and early-stage CRC with high sensitivity and specificity.

Gastroenterology · Sep 1, 2019Peer-reviewedCancer Biology

Spontaneous aggressive ERα+ mammary tumor model is driven by Kras activation

K. M. Campbell, K. A. O'Leary, D. E. Rugowski, W. A. Mulligan, E. K. Barnell, Z. L. Skidmore, K. Krysiak, M. Griffith, L. A. Schuler, O. L. Griffith

This study identifies Kras activation as a driver of spontaneous ERα+ mammary tumors in a murine model, validated through genomic and transcriptomic analyses.

bioRxiv · Oct 16, 2018Cancer Biology