Author

W. A. Mulligan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Approach:statistical model· 1 Research Focus:variant calling· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

D. E. Rugowski· 1 shared E. K. Barnell· 1 shared K. A. O'Leary· 1 shared K. Krysiak· 1 shared K. M. Campbell· 1 shared L. A. Schuler· 1 shared M. Griffith· 1 shared O. L. Griffith· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

D. E. Rugowski

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Spontaneous aggressive ERα+ mammary tumor model is driven by Kras activation

K. M. Campbell, K. A. O'Leary, D. E. Rugowski, W. A. Mulligan, E. K. Barnell, Z. L. Skidmore, K. Krysiak, M. Griffith, L. A. Schuler, O. L. Griffith

This study identifies Kras activation as a driver of spontaneous ERα+ mammary tumors in a murine model, validated through genomic and transcriptomic analyses.

bioRxiv · Oct 16, 2018Cancer Biology