Author

K. M. Campbell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

E. K. Barnell· 2 shared M. Griffith· 2 shared O. L. Griffith· 2 shared A. A. Chaudhuri· 1 shared A. Kalbasi· 1 shared A. R. Barnell· 1 shared A. Ribas· 1 shared D. E. Rugowski· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

E. K. Barnell

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Prioritization of SARS-CoV-2 epitopes using a pan-HLA and global population inference approach

K. M. Campbell, G. Steiner, D. K. Wells, A. Ribas, A. Kalbasi

The paper predicts SARS-CoV-2 peptide-HLA complexes across global populations and prioritizes epitopes for specific groups.

bioRxiv · Mar 30, 2020 · PreprintImmunology

Stool-derived eukaryotic RNA biomarkers for detection of high-risk adenomas

E. K. Barnell, Y. Kang, A. R. Barnell, K. M. Campbell, K. R. Kruse, E. M. Wurtzler, M. Griffith, A. A. Chaudhuri, O. L. Griffith

This study develops a stool-derived eukaryotic RNA biomarker model to detect high-risk adenomas and early-stage CRC with high sensitivity and specificity.

Gastroenterology · Sep 1, 2019Peer-reviewedCancer Biology

Spontaneous aggressive ERα+ mammary tumor model is driven by Kras activation

K. M. Campbell, K. A. O'Leary, D. E. Rugowski, W. A. Mulligan, E. K. Barnell, Z. L. Skidmore, K. Krysiak, M. Griffith, L. A. Schuler, O. L. Griffith

This study identifies Kras activation as a driver of spontaneous ERα+ mammary tumors in a murine model, validated through genomic and transcriptomic analyses.

bioRxiv · Oct 16, 2018Cancer Biology