Author

Eve Anderson

Institute of Genetics and CancerPrimary ORCID profile (0009-0002-8727-4639 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Eve AndersoniD1 paper · Cancer Research UK Scotland Institute

Biology Disciplines

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Developmental Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:ChIP-seq· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Laura A. LetticeiD· 2 shared Paul S. Devenney· 2 shared Robert E. HilliD· 2 shared Deborah Marie KurraschiD· 1 shared Fiona M. Kilanowski· 1 shared Iain WilliamsoniD· 1 shared Ilya M. FlyameriD· 1 shared Jeffrey J. SchoenebeckiD· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersDevelopmental Biology

Laura A. LetticeORCID

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2 papers

Recent·Most cited

A Highly Conserved Shh Enhancer Coordinates Hypothalamic and Craniofacial Development

Zoe Crane‐Smith, Jeffrey J. Schoenebeck, Katy A Graham, Paul S. Devenney, Lorraine Rose, Mark Ditzel, Eve Anderson, Joseph I. Thomson, Natasha Klenin, Deborah Marie Kurrasch, Laura A. Lettice, Robert E. Hill

This study shows that the conserved Shh enhancer SBE2 is critical for maintaining sufficient Shh expression in the rostral diencephalon, which is necessary for proper hypothalamic and craniofacial development.

Frontiers in Cell and Developmental Biology · Apr 1, 2021Peer-reviewedDevelopmental Biology

Developmentally regulated Shh expression is robust to TAD perturbations

Iain Williamson, Lauren Kane, Paul S. Devenney, Ilya M. Flyamer, Eve Anderson, Fiona M. Kilanowski, Robert E. Hill, Wendy A. Bickmore, Laura A. Lettice

The paper shows that Shh expression remains robust to TAD perturbations during development, except when enhancers are deleted.

Development · Sep 11, 2019Peer-reviewedDevelopmental Biology