Author

G. E. Pratt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

C. Ludwig· 1 shared J. Roberts· 1 shared M. A. C. Reed· 1 shared M. Hoogenkamp· 1 shared S. Al Omair· 1 shared S. Potluri· 1 shared S. Sarkar· 1 shared T. A. Perry· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

C. Ludwig

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1 paper

Recent·Most cited

T-ALL Proliferation Relies on Glutamine Uptake and EAAT1-dependent Conversion of Glutamine to Aspartate and Nucleotides

V. S. Stanulovic, S. Al Omair, M. A. C. Reed, S. Potluri, J. Roberts, T. A. Perry, S. Sarkar, G. E. Pratt, U. L. Gunther, C. Ludwig, M. Hoogenkamp

This study identifies EAAT1 as a critical target for T-ALL treatment by showing its role in glutamine conversion to nucleotides and aspartate essential for cell proliferation.

bioRxiv · Feb 8, 2020 · PreprintCancer Biology