Author

U. L. Gunther

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

S. Potluri· 2 shared A. Cunningham· 1 shared A. Erdem· 1 shared C. Ludwig· 1 shared E. Gonzalez· 1 shared F. Schnuetgen· 1 shared G. E. Pratt· 1 shared G. Papatzikas· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

S. Potluri

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Author profile →

2 papers

Recent·Most cited

Crosstalk between AML and stromal cells triggers acetate secretion through the metabolic rewiring of stromal cells

N. Vilaplana-Lopera, R. Almaghrabi, G. Papatzikas, V. Cuminetti, E. Gonzalez, M. Jeeves, A. Cunningham, A. Erdem, F. Schnuetgen, M. Raghavan, S. Potluri, J.-B. Cazier, J. J. Schuringa, M. A. Reed, L. Arranz, , P. Garcia

The paper identifies a metabolic crosstalk between AML and stromal cells where AML cells induce stromal cells to secrete acetate for their own energy needs.

bioRxiv · Jan 21, 2021Cell Biology

T-ALL Proliferation Relies on Glutamine Uptake and EAAT1-dependent Conversion of Glutamine to Aspartate and Nucleotides

V. S. Stanulovic, S. Al Omair, M. A. C. Reed, S. Potluri, J. Roberts, T. A. Perry, S. Sarkar, G. E. Pratt, U. L. Gunther, C. Ludwig, M. Hoogenkamp

This study identifies EAAT1 as a critical target for T-ALL treatment by showing its role in glutamine conversion to nucleotides and aspartate essential for cell proliferation.

bioRxiv · Feb 8, 2020 · PreprintCancer Biology