Author

M. Hoogenkamp

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:metabolomics· 1

Frequent Co-Authors

V. S. Stanulovic· 2 shared C. Ludwig· 1 shared D. G. Ward· 1 shared G. E. Pratt· 1 shared I. Dorrington· 1 shared J. Roberts· 1 shared M. A. C. Reed· 1 shared S. Al Omair· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

V. S. Stanulovic

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2 papers

Recent·Most cited

PHF6 Interacts with LMO2 During Normal Haematopoiesis and in Leukaemia and Regulates Gene Expression and Genome Integrity

V. S. Stanulovic, S. Binhassan, I. Dorrington, D. G. Ward, M. Hoogenkamp

This paper identifies PHF6 as a new LMO2 interacting factor that regulates gene expression and genome integrity during haematopoiesis and in T-ALL.

bioRxiv · Aug 18, 2020 · PreprintCancer Biology

T-ALL Proliferation Relies on Glutamine Uptake and EAAT1-dependent Conversion of Glutamine to Aspartate and Nucleotides

V. S. Stanulovic, S. Al Omair, M. A. C. Reed, S. Potluri, J. Roberts, T. A. Perry, S. Sarkar, G. E. Pratt, U. L. Gunther, C. Ludwig, M. Hoogenkamp

This study identifies EAAT1 as a critical target for T-ALL treatment by showing its role in glutamine conversion to nucleotides and aspartate essential for cell proliferation.

bioRxiv · Feb 8, 2020 · PreprintCancer Biology