Author

H. Billig

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:rat· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

J. Li· 2 shared J. Zhang· 2 shared L. R. Shao· 2 shared M. Brannstrom· 2 shared M. Hu· 2 shared P. Cui· 2 shared X. Li· 2 shared Y. Zhang· 2 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersPhysiology

J. Li

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2 papers

Recent·Most cited

Induction of hyperandrogenism and insulin resistance differentially modulates ferroptosis in uterine and placental tissues of pregnant rats

Y. Zhang, M. Hu, W. Jia, G. Liu, J. Zhang, B. Wang, J. Li, P. Cui, X. Li, S. Lager, A. N. Sferruzzi-Perri, Y. Han, S. Liu, X. Wu, M. Brannstrom, , H. Billig

This study investigates how maternal hyperandrogenism and insulin resistance in pregnant rats affect ferroptosis in uterine and placental tissues, revealing differential molecular and cellular mechanisms.

bioRxiv · Mar 30, 2020 · PreprintPhysiology

Uterine progesterone signaling is a target for metformin therapy in PCOS-like rats

M. Hu, Y. Zhang, J. Feng, X. Xu, J. Zhang, W. Zhao, X. Guo, J. Li, E. Vestin, P. Cui, X. Li, X.-k. Wu, M. Brannstrom, L. R. Shao, H. Billig

This study shows metformin improves uterine progesterone signaling in PCOS-like rats by reducing PGR and ER expression and correcting gene expression abnormalities.

bioRxiv · Mar 13, 2018Physiology