Author

Hyung Chul Lee

National Institutes of HealthPrimary ORCID profile (0000-0001-5438-1900 · 3 papers)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Hyung Chul LeeiD3 papers · University College London

Biology Disciplines

Click to filter author papers

Developmental Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Apratim MitraiD· 1 shared Beenish RahatiD· 1 shared Beth A. KozeliD· 1 shared Claudia M. GebertiD· 1 shared Connor M. KeaniD· 1 shared Danielle SpringeriD· 1 shared Jacob A. NoekeriD· 1 shared Karl E. PfeiferiD· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperDevelopmental Biology

Apratim MitraORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Cardiac pathologies in mouse loss of imprinting models are due to misexpression of H19 long noncoding RNA

Ki-Sun Park, Beenish Rahat, Hyung Chul Lee, Zu‐Xi Yu, Jacob A. Noeker, Apratim Mitra, Connor M. Kean, Russell H. Knutsen, Danielle Springer, Claudia M. Gebert, Beth A. Kozel, Karl E. Pfeifer

The paper shows that maternal loss of imprinting at the H19/IGF2 locus causes cardiac pathologies in mice, with Igf2 driving transient hypertrophy and H19 causing progressive fibrosis and reduced ventricular function.

eLife · Aug 17, 2021Peer-reviewedDevelopmental Biology