Author

I. E. Reider

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

C. C. Norbury· 2 shared N. J. Parekh· 2 shared T. E. Krouse· 2 shared A. M. Nelson· 1 shared B. M. Ward· 1 shared E. Lin· 1 shared R. P. Hobbs· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 17 mutual co-author links

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2 shared papersImmunology

C. C. Norbury

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2 papers

Recent·Most cited

γδ T cells mediate a requisite portion of a wound healing response triggered by cutaneous poxvirus infection

I. E. Reider, E. Lin, T. E. Krouse, N. J. Parekh, A. M. Nelson, C. C. Norbury

This study identifies γδ T cells as critical early mediators of a wound healing response during cutaneous poxvirus infection, independent of their role in virus replication control.

bioRxiv · Sep 1, 2020 · PreprintImmunology

Type I interferon-dependent CCL4 is induced by a cGAS/STING pathway that bypasses viral inhibition and protects infected tissue, independent of viral burden

N. J. Parekh, T. E. Krouse, I. E. Reider, R. P. Hobbs, B. M. Ward, C. C. Norbury

The paper shows that T1-IFN produced by a subset of VACV-infected cells via the cGAS/STING pathway protects mice by recruiting inflammatory monocytes, independent of viral replication.

bioRxiv · May 2, 2019Immunology