Author

I. Hilgendorf

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

R. Peyronnet· 2 shared A. Chan· 1 shared A. Faussner· 1 shared A. Janjic· 1 shared A. Klesen· 1 shared A. Lother· 1 shared A. Simon-Chica· 1 shared A. Yerdenova· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersBiophysics

R. Peyronnet

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Piezo1 stretch-activated channel activity differs between bone marrow-derived and cardiac tissue-resident macrophages

A. Simon-Chica, A. Klesen, R. Emig, A. Chan, D. Gruen, A. Lother, I. Hilgendorf, U. Ravens, P. Kohl, F. Schneider-Warme, R. Peyronnet

This study compares stretch-activated channel activity in bone marrow-derived and cardiac tissue-resident macrophages, finding that Piezo1 activity is absent in the latter.

bioRxiv · Nov 27, 2023 · PreprintBiophysics

Non-preferential, but detrimental accumulation of macrophages with clonal hematopoiesis-driver mutations in cardiovascular tissues

T.-s. Dederichs, A. Yerdenova, H. Horstmann, T. Vico, S. Nuebling, R. Peyronnet, C. von zur Muehlen, T. Heidt, D. Wolf, D. Westermann, I. Hilgendorf

The paper quantifies CHIP mutated macrophages in cardiovascular tissues and finds they do not preferentially accumulate but exhibit a more disease-prone phenotype.

bioRxiv · Sep 11, 2023 · PreprintImmunology

GPR55 in B cells limits atherosclerosis development and regulates plasma cell maturation

R. Guillamat-Prats, D. Hering, M. Rami, C. Haerdtner, D. Sanovito, P. Rinne, L. Bindila, M. Hristov, S. Pagano, N. Vuilleumier, S. Schmid, A. Janjic, W. Enard, C. Weber, L. Maegdefessel, , S. Steffens

This study shows that GPR55 in B cells reduces atherosclerosis by regulating plasma cell maturation and antibody production.

bioRxiv · Dec 20, 2021Immunology