Author

J. Craig

University of Lethbridge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Cancer Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Organism:human· 4 Approach:statistical model· 3 Assay:imaging· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

C. Haines· 2 shared Chelsea EkstrandiD· 2 shared K. Sullivan· 2 shared Keva Klamer· 2 shared A. Jorgensen· 1 shared A. Marson· 1 shared B. P. Koeleman· 1 shared C. Campbell· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

C. Haines

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Author profile →

4 papers

Recent·Most cited

Lower perceived stress enhances neural synchrony in perceptual and attentional cortices during naturalistic processing

J. Craig, Chelsea E. Matisz, Keva Klamer, C. Haines, K. Sullivan, Chelsea Ekstrand

The paper examines how perceived stress affects neural synchrony during naturalistic processing using fMRI data from the Naturalistic Neuroimaging Database.

bioRxiv · Sep 13, 2024 · PreprintNeuroscience

Trait level somatic arousal modulates fMRI neural synchrony to naturalistic stimuli

Keva Klamer, J. Craig, K. Sullivan, C. Haines, Chelsea Ekstrand

This study shows that higher baseline somatic arousal levels are linked to increased neural synchrony during naturalistic fMRI tasks.

bioRxiv · Sep 27, 2023 · PreprintNeuroscience

Polygenic risk score analysis reveals shared genetic burden between epilepsy and psychiatric comorbidities

C. Campbell, D. Lewis-Smith, C. Leu, H. Martins, S. Wolking, R. Krause, T. J. O'Brien, G. Sills, F. Zara, B. P. Koeleman, C. Depondt, A. Marson, H. Stefansson, K. Stefansson, J. Craig, , G. Cavalleri

This study uses polygenic risk scores to show that people with epilepsy have increased genetic risk for depression, psychosis, and ADHD, with stronger associations in drug-resistant cases.

medRxiv · Jul 4, 2023 · PreprintGenetic & Genomic Medicine

Loss of the Pioneer Factor FOXA1 Results in Genome-wide Epigenetic Reprogramming and activation of Interferon-Response Genes including CD274/PD-L1

W. Hu, H. Yamashita, J. Craig, V. Walter, J. I. Warrick, H. Al-Ahmadie, D. J. DeGraff

This study shows that FOXA1 loss in bladder cancer causes widespread epigenetic changes and activates interferon-response genes like PD-L1.

bioRxiv · Sep 11, 2020 · PreprintCancer Biology