Author

J. M. L. Ebos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

M. Dolan· 2 shared A. Dommer· 1 shared A. McKenery· 1 shared A. P. Arora· 1 shared A. Srinivasan· 1 shared C. Brackett· 1 shared D. Washington· 1 shared I. Puzanov· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

M. Dolan

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2 papers

Recent·Most cited

Robust eIF4B levels undermine invasive growth and immune evasion mechanisms in murine triple negative breast cancer models.

R. Smith, L. Z. D. Z. D. Diba, A. Srinivasan, R. A. Zollo, T. Ossevoort, Q. He, S. H. Colligan, M. Dolan, Y. Vedire, T. Sultana, M. Venkatesh, A. P. Arora, S. Gawlak, D. Washington, C. Brackett, , S. E. Walker

This study shows that higher eIF4B levels in murine triple negative breast cancer models are linked to less aggressive tumor growth and better patient outcomes.

bioRxiv · Sep 16, 2022 · PreprintCancer Biology

Acquired resistance to PD-L1 inhibition is associated with an enhanced type I IFN-stimulated secretory program in tumor cells

Y. Shi, M. Dolan, M. Mastri, A. McKenery, J. W. Hill, A. Dommer, S. Benzekry, M. Long, S. Abrams, I. Puzanov, J. M. L. Ebos

This study identifies an IFN-stimulated secretory program in PD-L1-resistant tumors that promotes immune evasion and shows that targeting this program can inhibit tumor growth.

bioRxiv · Jul 1, 2021 · PreprintCancer Biology