J. R. Naglik

This study identifies EGR1's role in regulating oral epithelial cell responses to C. albicans via the EGFR-ERK1/2 pathway, showing its involvement in immune response modulation.

This study shows candidalysin from C. albicans hyphae promotes both NETs and NLS, which together inhibit fungal growth and explain conflicting findings on ROS dependence in neutrophil responses.

This study identifies EGFR-MAPK adaptor proteins that mediate epithelial responses to C. albicans via candidalysin, showing their role in ERK1/2 signaling and cytokine production.

This paper investigates how Aspergillus fumigatus causes lung damage by analyzing host responses to different fungal morphotypes and secreted products in cultured cells and a mouse model.