Author

Jean‐Christophe Copin

École Polytechnique Fédérale de Lausanne

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 1

Frequent Co-Authors

Carmen SandiiD· 2 shared Hilal Ahmed LashueliD· 2 shared Johannes BurtscheriD· 2 shared Anass ChikiiD· 1 shared João RodriguesiD· 1 shared Marie-Isabelle Guillot de Suduiraut· 1 shared Senthil Kumar ThangarajiD· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

Carmen SandiORCID

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2 papers

Recent·Most cited

NeuroscienceeNeuroPeer-reviewedJun 2, 2020

Pronounced α-synuclein pathology in a seeding-based mouse model is not sufficient to induce mitochondrial respiration deficits in the striatum and amygdala

Johannes Burtscher, Jean‐Christophe Copin, Carmen Sandi, Hilal Ahmed Lashuel

This study shows that early α-synuclein pathology in a mouse model does not cause mitochondrial respiration deficits in the striatum and amygdala, even under corticosterone-induced hyperactivity.

11 citations

mouseproteomics

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 17, 2018

Chronic corticosterone enhancement aggravates alpha-synuclein brain spreading pathology and substantia nigra neurodegeneration in mice

Johannes Burtscher, Jean‐Christophe Copin, João Rodrigues…·Hilal Ahmed Lashuel

This study shows that chronic corticosterone worsens alpha-synuclein pathology and neurodegeneration in a mouse model of Parkinson's disease.

2 citations

mouseexperimental assay

𝕏 PostBluesky