SLX4IP acts in parallel to FANCM to limit BLM-dependent replication stress at ALT telomeres
Jessica Spindler, Francesca Pandolfo, Anna Eva Koch, Priscilla Piccirillo, Johanna I. Bihler, Marcel Morgenstern, Sandra Buschbaum, Joshua J. Coon, Robert Hänsel‐Hertsch, Kavi P.M. Mehta, Stephanie Panier
This study identifies SLX4IP as a suppressor of BLM-driven replication stress at ALT telomeres and reveals its parallel function with FANCM to regulate telomere maintenance.
bioRxiv · May 28, 2025 · PreprintCell Biology