Author

K. Calar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 3 Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

P. de la Puente· 3 shared H. Axemaker· 2 shared S. Plesselova· 2 shared C. Evans· 1 shared E. Sahly· 1 shared J. Wollman· 1 shared M. Jorgensen· 1 shared M. Petrasko· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 19 mutual co-author links

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3 shared papersCancer Biology

P. de la Puente

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Author profile →

3 papers

Recent·Most cited

Multicompartmentalized microvascularized tumor-on-a-chip to study tumor-stroma interactions and drug resistance in ovarian cancer

S. Plesselova, K. Calar, H. Axemaker, E. Sahly, P. de la Puente

This paper develops a tumor-on-a-chip model to study ovarian cancer drug resistance by mimicking tumor-stroma interactions and compartmentalized microenvironment.

bioRxiv · May 29, 2024Bioengineering

Normal Uterine Fibroblast Are Reprogramed into Ovarian Cancer-Associated Fibroblasts by Ovarian Tumor-derived Conditioned Media

H. Axemaker, S. Plesselova, K. Calar, M. Jorgensen, J. Wollman, P. de la Puente

This study reprograms normal uterine fibroblasts into ovarian cancer-associated fibroblasts using ovarian tumor-derived conditioned media, demonstrating enhanced tumor progression and drug resistance.

bioRxiv · Sep 29, 2023 · PreprintCancer Biology

Bioengineering a novel 3D in-vitro model to recreate physiological oxygen levels and tumor-immune interactions

S. Bhattacharya, K. Calar, C. Evans, M. Petrasko, P. de la Puente

This study develops a 3D in vitro model (3D-O) that mimics physiological oxygen levels in breast cancer, showing hypoxia-induced changes that promote immune evasion and reduced CD8+ T cell infiltration.

bioRxiv · Nov 1, 2019Cancer Biology