Author

K. Mueller-Decker

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

A. H. C. van Kampen· 1 shared A. Hill· 1 shared A. Jauch· 1 shared A. Kraemer· 1 shared A. Noronha· 1 shared A. Patrizi· 1 shared A. T. Moussa· 1 shared B. Kraft· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. H. C. van Kampen

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2 papers

Recent·Most cited

STIL overexpression shortens lifespan and reduces tumor formation in mice

A. T. Moussa, M. R. Cosenza, T. Wohlfromm, K. Brobeil, A. Hill, A. Patrizi, K. Mueller-Decker, T. Holland-Letz, A. Jauch, B. Kraft, A. Kraemer

This study shows that STIL overexpression increases centrosome numbers, leading to aneuploidy and reduced tumor formation and lifespan in mice.

bioRxiv · Dec 4, 2023Cancer Biology

Time-resolved profiling reveals ATF3 as a novel mediator of endocrine resistance in breast cancer

S. Borgoni, E. Sofyali, M. Soleimani Dodaran, H. Wilhelm, K. Mueller-Decker, R. Will, A. Noronha, P. J. Verschure, Y. Yarden, L. Magnani, A. H. C. van Kampen, P. D. Moerland, S. Wiemann

This study identifies ATF3 as a novel mediator of endocrine resistance in breast cancer through time-resolved profiling and validates its role in key signaling pathways and clinical relevance.

bioRxiv · Jun 8, 2020Cancer Biology