Author

Ke Hu

Beihang University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

Anqiang SuniD· 1 shared Christopher Bare· 1 shared Claudia GiambartolomeiiD· 1 shared Diego GalloiD· 1 shared Eiji Nagayasu· 1 shared Joel T Dudley· 1 shared John F Fullard· 1 shared John Murray· 1 shared

Activity by Year

2017-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperBioengineering

Anqiang SunORCID

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3 papers

Recent·Most cited

A vascular chip for disease-relevant flow shear stress topology

Kexin Li, S M Yang, Ke Hu, Zhuqing Liang, Xing Zhang, Junjie Yang, Umberto Morbiducci, Valentina Mazzi, Diego Gallo, Li Wang, Min Wang, Xiaoning Sun, Zengsheng Chen, Anqiang Sun, Lingqian Chang, , Xiao Liu

This paper introduces a vascular chip that enables controlled experimentation with disease-relevant wall shear stress topology, revealing its effects on endothelial mechanics and function.

bioRxiv · Jul 7, 2026 · PreprintBioengineering

Cell-type specific open chromatin profiling in human postmortem brain infers functional roles for non-coding schizophrenia loci

John F Fullard, Claudia Giambartolomei, Mads E Hauberg, Ke Hu, Christopher Bare, Joel T Dudley, Manuel Mattheisen, Joseph K Pickrell, Vahram Haroutunian, Panos Roussos

The paper uses ATAC-seq to profile open chromatin in human brain cells, identifying cell-type-specific regulatory elements and linking them to schizophrenia risk variants.

Human Molecular Genetics · Mar 14, 2017Peer-reviewedGenomics

Loss of a doublecortin (DCX) domain containing protein causes structural defects in a tubulin-based organelle of Toxoplasma gondii and impairs host cell invasion.

Eiji Nagayasu, Yu-chen Hwang, Jun Liu, John Murray, Ke Hu

This study shows that TgDCX is essential for the structural integrity of the conoid in Toxoplasma gondii and its loss impairs host cell invasion.

Molecular Biology of the Cell · Feb 1, 2017Peer-reviewedCell Biology