Kevyn R. Martins

This study identifies Oncostatin M (OSM) as a critical regulator of pulmonary host responses during influenza and bacterial pneumonia, revealing non-canonical signaling pathways.

This study identifies pathogen-specific neutrophil surface protein phenotypes in bacterial pneumonia, revealing tissue- and infection-type dependent immune responses.

The paper shows that neutrophils in a murine pneumococcal pneumonia model transition through two distinct transcriptional trajectories with associated metabolic changes.