Author

L. Fernandez-Mosquera

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

N. Raimundo· 2 shared A. Mishra· 1 shared C. Muehle· 1 shared D. Feigenbutz· 1 shared D. Hornburg· 1 shared E. Ikonen· 1 shared F. Meissner· 1 shared I. Dudanova· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

N. Raimundo

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Amyloid-like aggregates cause lysosomal defects in neurons via gain-of-function toxicity

I. Riera-Tur, T. Schaefer, D. Hornburg, A. Mishra, L. Fernandez-Mosquera, D. Feigenbutz, P. Auer, M. Mann, W. Baumeister, R. Klein, F. Meissner, N. Raimundo, R. Fernandez-Busnadiego, I. Dudanova

This study shows that amyloid-like aggregates impair lysosomal function in neurons by sequestering AP-31, leading to neurotoxicity.

bioRxiv · Dec 16, 2019 · PreprintCell Biology

Mitochondrial biogenesis is transcriptionally repressed in lysosomal lipid storage diseases

K. F. Yambire, L. Fernandez-Mosquera, R. Steinfeld, C. Muehle, E. Ikonen, I. Milosevic, N. Raimundo

This study identifies a transcriptional repression of mitochondrial biogenesis in lysosomal storage diseases through KLF2 and ETV1, showing their roles in disease mechanisms.

bioRxiv · Feb 6, 2019Cell Biology